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Its inhibition leads to a significant reduction in IgM production, highlighting the crucial role of SLC1A5 as a transportation protein in orchestrating the intricate process of immunoglobulin generation within B cells [121]

Similarly, NSC34 motor neuron-like cells stably expressing the human G93A mutant form of SOD1 displayed a significant and selective depletion of mitochondrial GSH content in comparison to parental cells, reminiscent of some forms of familial ALS [16]