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At the cellular level, OA is the result of an imbalance between anabolic and catabolic processes

DSIP has been studied in neurological contexts, but that does not mean it improves brain function in healthy people

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These in vitro results have been further validated in murine models of malaria infection and rheumatoid arthritis, where G6PD-d or inhibition leads to less severe proinflammatory responses to stimulation marked by reduced Th1 and Th17 differentiation, decreased proatherogenic cytokines IL-1, IL-6, IL-12, and TNF, and no impact on IL-10 and TGF (classically Treg-derived) 120,121,122,123,124